December 2014
Volume 13Issue 12p3224-3715
Open Access
On the cover: O-GlcNAc modification of the master osteogenic transcriptional regulator Runx2 [runt-related transcription factor 2/core-binding factor subunit α 1 (Cbfa1)] reveals new
links between glucose homeostasis and bone formation. Higher energy collisional dissociation
(HCD)-generated HexNAc product ions were utilized to trigger acquisition of electron
transfer dissociation (ETD) fragmentation spectra to facilitate characterization of
O-GlcNAc modified sites. For details, see the article by Alexis K. Nagel, et al., pages 3381–3395....Show more
On the cover: O-GlcNAc modification of the master osteogenic transcriptional regulator Runx2 [runt-related transcription factor 2/core-binding factor subunit α 1 (Cbfa1)] reveals new
links between glucose homeostasis and bone formation. Higher energy collisional dissociation
(HCD)-generated HexNAc product ions were utilized to trigger acquisition of electron
transfer dissociation (ETD) fragmentation spectra to facilitate characterization of
O-GlcNAc modified sites. For details, see the article by Alexis K. Nagel, et al., pages 3381–3395.
![On the cover: <em>O</em>-GlcNAc modification of the master osteogenic transcriptional regulator Runx2 [<em>runt</em>-related transcription factor 2/core-binding factor subunit α 1 (Cbfa1)] reveals new
links between glucose homeostasis and bone formation. Higher energy collisional dissociation
(HCD)-generated HexNAc product ions were utilized to trigger acquisition of electron
transfer dissociation (ETD) fragmentation spectra to facilitate characterization of
<em>O</em>-GlcNAc modified sites. For details, see the article by Alexis K. Nagel, <em>et al.</em>, pages <a href="http://dx.doi.org/10.1074/mcp.M114.040691" target="_blank">3381–3395</a>.](/cms/asset/atypon:cms:attachment:img:d18e6:rev:1616179761875-788:pii:S1535947620X61858/cover.tif.jpg)
